Youtube daily report w Dec 26 2016

Neurons are the cells that make up our nervous system, and they're made up of three main

parts.

The dendrites, which are little branches off of the neuron that receive signals from other

neurons, the soma, or cell body, which has all of the neuron's main organelles like

the nucleus, and the axon which is intermittently wrapped in fatty myelin.

Those dendrites receive signals from other neurons via neurotransmitters, which when

they bind to receptors on the dendrite act as a chemical signal.

That binding opens ion channels that allow charged ions to flow in and out of the cell,

converting the chemical signal into an electrical signal.

Since a single neuron can have a ton of dendrites receiving input, if the combined effect of

multiple dendrites changes the overall charge of the cell enough, then it triggers an action

potential- which is an electrical signal that races down the axon up to 100 meters per second,

triggering the release of neurotransmitter on the other end and further relaying the

signal.

So neurons use neurotransmitters as a signal to communicate with each other, but they use

the action potential to propagate that signal within the cell.

Some of these neurons can be very long, especially ones that go from the spinal cord to the toes,

so the movement of this electrical signal is super important!

But why does the cell have an electric charge in the first place?

Well, it's based on the different concentrations of ions on the inside versus outside of the

cell.

Generally speaking, there are more Na+ or sodium ions, Cl- or chloride ions, and Ca2+

or calcium ions on the outside, and more K+ or potassium ion and A- which we just use

for negatively charged anions, on the inside.

Overall, the distribution of these ions gives the cell a net negative charge of close to

-65 millivolts relative to the outside environment - this is called the neuron's resting membrane

potential.

When a neurotransmitter binds to a receptor on the dendrite, a ligand-gated ion channel

opens up to allow certain ions to flow in, depending on the channel.

Ligand-gated literally means that the gate responds to a ligand, which in this case is

a neurotransmitter.

So if we take the example of a ligand-gated Na+ ion channel, which, when it opens, lets

Na+ flow into the cell.

The extra positive charge that flows in makes the cell less negative (since remember it's

usually -65mV), and therefore less "polar" - so that's why gaining positive charge

is called depolarization.

Neurotransmitters typically open various ligand-gated ion channels all at once, so ions like sodium

and calcium, may flow in, while other ions like potassium, may flow out, which would

actually mean some positive charge leaves the cell.

In the end though - when it's all added up - if there is a net influx of positive

charge, then it's called an excitatory postsynaptic potential (EPSP).

In contrast, the opening of only ligand-gated Cl- ion channels would cause a net influx

of negative charge, creating an inhibitory postsynaptic potential (IPSP), making the

cell potential more negative or repolarizing it.

Now, a single EPSP or IPSP causes only a small change on the resting membrane potential,

but, if there are enough EPSPs across multiple sites on the dendrites then collectively they

can push the membrane potential to a specific threshold value- typically about -55mV, although

this can vary by tissue.

When this occurs, it triggers the opening of voltage-gated Na+ channels at the start

of the axon - the axon hillock, voltage-gated channels open in response to a change in voltage,

and when these open sodium to rush into the cell.

The influx of sodium ions and the resulting change in membrane potential causes nearby

voltage-gated sodium channels to open up as well - setting off a chain reaction that continues

down the entire length of the axon—which is our action potential, and when this happens,

we say that the neuron has 'fired.'

Once a lot of sodium has rushed across the neuronal membrane, the call actually becomes

positively charged relative to the external environment - up to about +40mV.

The depolarization process ends when the sodium channel stops allowing sodium to flow into

the cells- a process known as inactivation.

But this state is different than when the channel's closed, or open for that matter,

which is what most of the other channels have.

The voltage-gated sodium channel, though, is unique in that it has what's known as

the inactivation gate, which blocks sodium influx shortly after depolarization, until

the cell repolarizes and the channel enters the closed state again and the inactivation

gate stops blocking influx, although even though the inactivation gate's not blocking,

the channel's still closed so no sodium enters the cell.

This middle open state therefore is the only state where sodium gets let into the cell

through the channel, and this is a very short window of time.

Now in addition to these sodium voltage-gated channels, we've also got potassium voltage-gated

channels, which are slow to respond and don't open until the sodium channels have already

opened and become inactivated.

The result is that after the initial sodium rush into the cell, potassium flows out of

the cell down its own electrochemical gradient- removing some positive charge and blunting

the effect of the sodium depolarization.

The potassium channels, do not have a separate inactivation gate and therefore remain open

for slightly longer, which means that there is a period of time when there is a net movement

of positive ions out of the cell, causing the membrane potential to become more negative,

or repolarize.

During this repolarization phase, the cell also relies on the sodium-potassium pump,

an active transporter that moves three sodiums out of the cell and two potassiums into it.

It's during this repolarization phase that the cell's in its absolute refractory period,

since the sodium channels are inactivated and won't respond to any amount of stimuli.

This absolute refractory period keeps the action potentials from happening too close

together in time, but also keeps the action potential moving in one direction.

The combined efforts of this pump and the extended opening of the potassium channels

results in a small period of overcorrection where the neuron becomes hyperpolarized relative

to the resting potential, and at this point the sodium channels go back to their initial

closed state, and for a short period the potassium channels stay open.

Now we're in the relative refractory period since the sodium channels are closed but can

be activated, but because the potassium channels are still open and we're in a hyperpolarized

state, so it's takes a strong stimulus to do so.

Finally, as the potassium channels close, the neuron returns to it's resting membrane

potential.

Alright, as a quick graphical recap, with membrane potential on the y and time on the

x.

First we start at resting potential of around -65 mV and voltage-gated sodium and potassium

channels are closed, we receive EPSPs enough to hit threshold at about -55 mV, voltage-gated

sodium channels open and we reach a peak of about +40 mV, at which point the sodium channels

become inactivated and we're in the absolute refractory period.

Voltage-gated potassium channels open, and along with the sodium-potassium pump, start

to repolarize the cell, so much so that it overshoots and hyperpolarizes the cell.

Next the sodium channels enter their closed resting state as potassium channels start

to close we're in the relative refractory period, until finally they all close and we

reach our resting membrane potential.

Alright, so this process of positive sodium ions moving in and depolarizing the cell transmits

the electrical signal down the length of the axon.

Great.

But really, this process isn't that fast.

So that's where the fatty myelin comes in, which comes from glial cells like Schwann

cells or oligodendrocytes.

These myelinated areas don't have voltage-gated ion channels spanning the membrane, so ions

can't simply flow into the cell, that only happens in the spots between the myelin, called

nodes of Ranvier.

So instead of propagating via channels, the charge essentially jumps from node to node.

That said though, these ions aren't just diffusing down the length of the myelin to

the other side...that'd be way to slow.

What actually happens is more like the sodium ions rushing in bumps other positive sodium

ions already inside the cell, which bumps another one, and so on until it reaches the

next node.

The charge moving in this way with the myelinated areas moves really fast, and is called saltatory

conduction, which makes it look like the action potential "jumps" from one one node to

the next.

Okay extremely quick recap - neuron action potentials happen when dendrites receive enough

EPSPs to open voltage-gated sodium channels, which cause rapid depolarization of the neuronal

membrane and propagation of an electrical charge from node to node down the length of

the axon.

Thanks for watching, you can help support us by donating on patreon, or subscribing

to our channel, or telling your friends about us on social media

For more infomation >> Neuron action potential - physiology - Duration: 10:25.

-------------------------------------------

Ikv 103. Шведская ветка. Это УЖАС - Убил всех. 12 фрагов. 🇸🇪 🔝 World Of Tanks ( wot ) - Duration: 10:02.

For more infomation >> Ikv 103. Шведская ветка. Это УЖАС - Убил всех. 12 фрагов. 🇸🇪 🔝 World Of Tanks ( wot ) - Duration: 10:02.

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ThatTutorGuy.com

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Como se planejar para ter um ano equilibrado e rentável - Duration: 59:50.

For more infomation >> Como se planejar para ter um ano equilibrado e rentável - Duration: 59:50.

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Como prevenir que seu site seja hackeado - SECNET - Duration: 2:40.

For more infomation >> Como prevenir que seu site seja hackeado - SECNET - Duration: 2:40.

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ricos vs pobre - Duration: 3:49.

For more infomation >> ricos vs pobre - Duration: 3:49.

-------------------------------------------

Neuron action potential - physiology - Duration: 10:25.

Neurons are the cells that make up our nervous system, and they're made up of three main

parts.

The dendrites, which are little branches off of the neuron that receive signals from other

neurons, the soma, or cell body, which has all of the neuron's main organelles like

the nucleus, and the axon which is intermittently wrapped in fatty myelin.

Those dendrites receive signals from other neurons via neurotransmitters, which when

they bind to receptors on the dendrite act as a chemical signal.

That binding opens ion channels that allow charged ions to flow in and out of the cell,

converting the chemical signal into an electrical signal.

Since a single neuron can have a ton of dendrites receiving input, if the combined effect of

multiple dendrites changes the overall charge of the cell enough, then it triggers an action

potential- which is an electrical signal that races down the axon up to 100 meters per second,

triggering the release of neurotransmitter on the other end and further relaying the

signal.

So neurons use neurotransmitters as a signal to communicate with each other, but they use

the action potential to propagate that signal within the cell.

Some of these neurons can be very long, especially ones that go from the spinal cord to the toes,

so the movement of this electrical signal is super important!

But why does the cell have an electric charge in the first place?

Well, it's based on the different concentrations of ions on the inside versus outside of the

cell.

Generally speaking, there are more Na+ or sodium ions, Cl- or chloride ions, and Ca2+

or calcium ions on the outside, and more K+ or potassium ion and A- which we just use

for negatively charged anions, on the inside.

Overall, the distribution of these ions gives the cell a net negative charge of close to

-65 millivolts relative to the outside environment - this is called the neuron's resting membrane

potential.

When a neurotransmitter binds to a receptor on the dendrite, a ligand-gated ion channel

opens up to allow certain ions to flow in, depending on the channel.

Ligand-gated literally means that the gate responds to a ligand, which in this case is

a neurotransmitter.

So if we take the example of a ligand-gated Na+ ion channel, which, when it opens, lets

Na+ flow into the cell.

The extra positive charge that flows in makes the cell less negative (since remember it's

usually -65mV), and therefore less "polar" - so that's why gaining positive charge

is called depolarization.

Neurotransmitters typically open various ligand-gated ion channels all at once, so ions like sodium

and calcium, may flow in, while other ions like potassium, may flow out, which would

actually mean some positive charge leaves the cell.

In the end though - when it's all added up - if there is a net influx of positive

charge, then it's called an excitatory postsynaptic potential (EPSP).

In contrast, the opening of only ligand-gated Cl- ion channels would cause a net influx

of negative charge, creating an inhibitory postsynaptic potential (IPSP), making the

cell potential more negative or repolarizing it.

Now, a single EPSP or IPSP causes only a small change on the resting membrane potential,

but, if there are enough EPSPs across multiple sites on the dendrites then collectively they

can push the membrane potential to a specific threshold value- typically about -55mV, although

this can vary by tissue.

When this occurs, it triggers the opening of voltage-gated Na+ channels at the start

of the axon - the axon hillock, voltage-gated channels open in response to a change in voltage,

and when these open sodium to rush into the cell.

The influx of sodium ions and the resulting change in membrane potential causes nearby

voltage-gated sodium channels to open up as well - setting off a chain reaction that continues

down the entire length of the axon—which is our action potential, and when this happens,

we say that the neuron has 'fired.'

Once a lot of sodium has rushed across the neuronal membrane, the call actually becomes

positively charged relative to the external environment - up to about +40mV.

The depolarization process ends when the sodium channel stops allowing sodium to flow into

the cells- a process known as inactivation.

But this state is different than when the channel's closed, or open for that matter,

which is what most of the other channels have.

The voltage-gated sodium channel, though, is unique in that it has what's known as

the inactivation gate, which blocks sodium influx shortly after depolarization, until

the cell repolarizes and the channel enters the closed state again and the inactivation

gate stops blocking influx, although even though the inactivation gate's not blocking,

the channel's still closed so no sodium enters the cell.

This middle open state therefore is the only state where sodium gets let into the cell

through the channel, and this is a very short window of time.

Now in addition to these sodium voltage-gated channels, we've also got potassium voltage-gated

channels, which are slow to respond and don't open until the sodium channels have already

opened and become inactivated.

The result is that after the initial sodium rush into the cell, potassium flows out of

the cell down its own electrochemical gradient- removing some positive charge and blunting

the effect of the sodium depolarization.

The potassium channels, do not have a separate inactivation gate and therefore remain open

for slightly longer, which means that there is a period of time when there is a net movement

of positive ions out of the cell, causing the membrane potential to become more negative,

or repolarize.

During this repolarization phase, the cell also relies on the sodium-potassium pump,

an active transporter that moves three sodiums out of the cell and two potassiums into it.

It's during this repolarization phase that the cell's in its absolute refractory period,

since the sodium channels are inactivated and won't respond to any amount of stimuli.

This absolute refractory period keeps the action potentials from happening too close

together in time, but also keeps the action potential moving in one direction.

The combined efforts of this pump and the extended opening of the potassium channels

results in a small period of overcorrection where the neuron becomes hyperpolarized relative

to the resting potential, and at this point the sodium channels go back to their initial

closed state, and for a short period the potassium channels stay open.

Now we're in the relative refractory period since the sodium channels are closed but can

be activated, but because the potassium channels are still open and we're in a hyperpolarized

state, so it's takes a strong stimulus to do so.

Finally, as the potassium channels close, the neuron returns to it's resting membrane

potential.

Alright, as a quick graphical recap, with membrane potential on the y and time on the

x.

First we start at resting potential of around -65 mV and voltage-gated sodium and potassium

channels are closed, we receive EPSPs enough to hit threshold at about -55 mV, voltage-gated

sodium channels open and we reach a peak of about +40 mV, at which point the sodium channels

become inactivated and we're in the absolute refractory period.

Voltage-gated potassium channels open, and along with the sodium-potassium pump, start

to repolarize the cell, so much so that it overshoots and hyperpolarizes the cell.

Next the sodium channels enter their closed resting state as potassium channels start

to close we're in the relative refractory period, until finally they all close and we

reach our resting membrane potential.

Alright, so this process of positive sodium ions moving in and depolarizing the cell transmits

the electrical signal down the length of the axon.

Great.

But really, this process isn't that fast.

So that's where the fatty myelin comes in, which comes from glial cells like Schwann

cells or oligodendrocytes.

These myelinated areas don't have voltage-gated ion channels spanning the membrane, so ions

can't simply flow into the cell, that only happens in the spots between the myelin, called

nodes of Ranvier.

So instead of propagating via channels, the charge essentially jumps from node to node.

That said though, these ions aren't just diffusing down the length of the myelin to

the other side...that'd be way to slow.

What actually happens is more like the sodium ions rushing in bumps other positive sodium

ions already inside the cell, which bumps another one, and so on until it reaches the

next node.

The charge moving in this way with the myelinated areas moves really fast, and is called saltatory

conduction, which makes it look like the action potential "jumps" from one one node to

the next.

Okay extremely quick recap - neuron action potentials happen when dendrites receive enough

EPSPs to open voltage-gated sodium channels, which cause rapid depolarization of the neuronal

membrane and propagation of an electrical charge from node to node down the length of

the axon.

Thanks for watching, you can help support us by donating on patreon, or subscribing

to our channel, or telling your friends about us on social media

For more infomation >> Neuron action potential - physiology - Duration: 10:25.

-------------------------------------------

Quaker® Oats Smoothie

For more infomation >> Quaker® Oats Smoothie

-------------------------------------------

Neuron action potential - physiology - Duration: 10:25.

Neurons are the cells that make up our nervous system, and they're made up of three main

parts.

The dendrites, which are little branches off of the neuron that receive signals from other

neurons, the soma, or cell body, which has all of the neuron's main organelles like

the nucleus, and the axon which is intermittently wrapped in fatty myelin.

Those dendrites receive signals from other neurons via neurotransmitters, which when

they bind to receptors on the dendrite act as a chemical signal.

That binding opens ion channels that allow charged ions to flow in and out of the cell,

converting the chemical signal into an electrical signal.

Since a single neuron can have a ton of dendrites receiving input, if the combined effect of

multiple dendrites changes the overall charge of the cell enough, then it triggers an action

potential- which is an electrical signal that races down the axon up to 100 meters per second,

triggering the release of neurotransmitter on the other end and further relaying the

signal.

So neurons use neurotransmitters as a signal to communicate with each other, but they use

the action potential to propagate that signal within the cell.

Some of these neurons can be very long, especially ones that go from the spinal cord to the toes,

so the movement of this electrical signal is super important!

But why does the cell have an electric charge in the first place?

Well, it's based on the different concentrations of ions on the inside versus outside of the

cell.

Generally speaking, there are more Na+ or sodium ions, Cl- or chloride ions, and Ca2+

or calcium ions on the outside, and more K+ or potassium ion and A- which we just use

for negatively charged anions, on the inside.

Overall, the distribution of these ions gives the cell a net negative charge of close to

-65 millivolts relative to the outside environment - this is called the neuron's resting membrane

potential.

When a neurotransmitter binds to a receptor on the dendrite, a ligand-gated ion channel

opens up to allow certain ions to flow in, depending on the channel.

Ligand-gated literally means that the gate responds to a ligand, which in this case is

a neurotransmitter.

So if we take the example of a ligand-gated Na+ ion channel, which, when it opens, lets

Na+ flow into the cell.

The extra positive charge that flows in makes the cell less negative (since remember it's

usually -65mV), and therefore less "polar" - so that's why gaining positive charge

is called depolarization.

Neurotransmitters typically open various ligand-gated ion channels all at once, so ions like sodium

and calcium, may flow in, while other ions like potassium, may flow out, which would

actually mean some positive charge leaves the cell.

In the end though - when it's all added up - if there is a net influx of positive

charge, then it's called an excitatory postsynaptic potential (EPSP).

In contrast, the opening of only ligand-gated Cl- ion channels would cause a net influx

of negative charge, creating an inhibitory postsynaptic potential (IPSP), making the

cell potential more negative or repolarizing it.

Now, a single EPSP or IPSP causes only a small change on the resting membrane potential,

but, if there are enough EPSPs across multiple sites on the dendrites then collectively they

can push the membrane potential to a specific threshold value- typically about -55mV, although

this can vary by tissue.

When this occurs, it triggers the opening of voltage-gated Na+ channels at the start

of the axon - the axon hillock, voltage-gated channels open in response to a change in voltage,

and when these open sodium to rush into the cell.

The influx of sodium ions and the resulting change in membrane potential causes nearby

voltage-gated sodium channels to open up as well - setting off a chain reaction that continues

down the entire length of the axon—which is our action potential, and when this happens,

we say that the neuron has 'fired.'

Once a lot of sodium has rushed across the neuronal membrane, the call actually becomes

positively charged relative to the external environment - up to about +40mV.

The depolarization process ends when the sodium channel stops allowing sodium to flow into

the cells- a process known as inactivation.

But this state is different than when the channel's closed, or open for that matter,

which is what most of the other channels have.

The voltage-gated sodium channel, though, is unique in that it has what's known as

the inactivation gate, which blocks sodium influx shortly after depolarization, until

the cell repolarizes and the channel enters the closed state again and the inactivation

gate stops blocking influx, although even though the inactivation gate's not blocking,

the channel's still closed so no sodium enters the cell.

This middle open state therefore is the only state where sodium gets let into the cell

through the channel, and this is a very short window of time.

Now in addition to these sodium voltage-gated channels, we've also got potassium voltage-gated

channels, which are slow to respond and don't open until the sodium channels have already

opened and become inactivated.

The result is that after the initial sodium rush into the cell, potassium flows out of

the cell down its own electrochemical gradient- removing some positive charge and blunting

the effect of the sodium depolarization.

The potassium channels, do not have a separate inactivation gate and therefore remain open

for slightly longer, which means that there is a period of time when there is a net movement

of positive ions out of the cell, causing the membrane potential to become more negative,

or repolarize.

During this repolarization phase, the cell also relies on the sodium-potassium pump,

an active transporter that moves three sodiums out of the cell and two potassiums into it.

It's during this repolarization phase that the cell's in its absolute refractory period,

since the sodium channels are inactivated and won't respond to any amount of stimuli.

This absolute refractory period keeps the action potentials from happening too close

together in time, but also keeps the action potential moving in one direction.

The combined efforts of this pump and the extended opening of the potassium channels

results in a small period of overcorrection where the neuron becomes hyperpolarized relative

to the resting potential, and at this point the sodium channels go back to their initial

closed state, and for a short period the potassium channels stay open.

Now we're in the relative refractory period since the sodium channels are closed but can

be activated, but because the potassium channels are still open and we're in a hyperpolarized

state, so it's takes a strong stimulus to do so.

Finally, as the potassium channels close, the neuron returns to it's resting membrane

potential.

Alright, as a quick graphical recap, with membrane potential on the y and time on the

x.

First we start at resting potential of around -65 mV and voltage-gated sodium and potassium

channels are closed, we receive EPSPs enough to hit threshold at about -55 mV, voltage-gated

sodium channels open and we reach a peak of about +40 mV, at which point the sodium channels

become inactivated and we're in the absolute refractory period.

Voltage-gated potassium channels open, and along with the sodium-potassium pump, start

to repolarize the cell, so much so that it overshoots and hyperpolarizes the cell.

Next the sodium channels enter their closed resting state as potassium channels start

to close we're in the relative refractory period, until finally they all close and we

reach our resting membrane potential.

Alright, so this process of positive sodium ions moving in and depolarizing the cell transmits

the electrical signal down the length of the axon.

Great.

But really, this process isn't that fast.

So that's where the fatty myelin comes in, which comes from glial cells like Schwann

cells or oligodendrocytes.

These myelinated areas don't have voltage-gated ion channels spanning the membrane, so ions

can't simply flow into the cell, that only happens in the spots between the myelin, called

nodes of Ranvier.

So instead of propagating via channels, the charge essentially jumps from node to node.

That said though, these ions aren't just diffusing down the length of the myelin to

the other side...that'd be way to slow.

What actually happens is more like the sodium ions rushing in bumps other positive sodium

ions already inside the cell, which bumps another one, and so on until it reaches the

next node.

The charge moving in this way with the myelinated areas moves really fast, and is called saltatory

conduction, which makes it look like the action potential "jumps" from one one node to

the next.

Okay extremely quick recap - neuron action potentials happen when dendrites receive enough

EPSPs to open voltage-gated sodium channels, which cause rapid depolarization of the neuronal

membrane and propagation of an electrical charge from node to node down the length of

the axon.

Thanks for watching, you can help support us by donating on patreon, or subscribing

to our channel, or telling your friends about us on social media

For more infomation >> Neuron action potential - physiology - Duration: 10:25.

-------------------------------------------

Ikv 103. Шведская ветка. Это УЖАС - Убил всех. 12 фрагов. 🇸🇪 🔝 World Of Tanks ( wot ) - Duration: 10:02.

For more infomation >> Ikv 103. Шведская ветка. Это УЖАС - Убил всех. 12 фрагов. 🇸🇪 🔝 World Of Tanks ( wot ) - Duration: 10:02.

-------------------------------------------

'La La Land' Trailer

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Keys of Christmas

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MEN | How You See Me - Duration: 4:17.

For more infomation >> MEN | How You See Me - Duration: 4:17.

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Warframe - How to Get Nidas Parts ASAP - Update 19.5 The Glast Gambit [1080HD] - Duration: 1:59.

Ahoy thar, Legion!

Hope the holiday weekend treated ya well!

I'm actually recordin' this in between batches a-cookies so I hope ya don't mind if I keep

this Nidus farmin' guide quick n' concise.

In order to craft the infested caster, you'll need to have completed The War Within questline

as well as The Glast Gambit,

as one grants you a copy of his blueprint

and the other allows ya access to Kuva which ya need to combine em all together

But!

Ya don't need to have completed either to farm up the other three parts!

Which uh... most would say takes the longest.

All ya gotta do is head off to Oestrus on Eris and run the newish Infested Salvage mode!

The process is pretty simple, kill all the things,

loot the blue glowin' blocks that drop,

and use them on the objective markers to make keep the bubbles big.

Keepin' em big makes sure the nearby console stays covered and extractin' information.

There's three objectives to keep in mind,

and the more you have operational the faster the round ticks by so you'll want all of em

runnin' at max capacity because we got ourselves a Warframe to grind!

The parts themselves only have a chance to appear on the fourth round of rewards,

So, again, yer gonna need to stay through four rewards before you have a shot at em.

but if you can hold out that long and survive till the end, you're gonna have this to look forward to!

Notice I didn't get a Nidus part.

Odds are ya probably won't either haha

Sorry, laughter helps dull the pain.

But!

If you do these missions enough, you *will* get em to drop.

Eventually.

Again the blueprint is still tied to the Glast Gambit quest,

which personally I'm waiting for them to fix before I even pick it up,

but these snickerdoodles ain't gonna make themselves so with that

I wish ya good luck on your dice rolls, Legion!

Headin' back to the cookie factory formation.

Think I'mma decorate one to look like Ordis.

Just so I can eat it and taunt the Misses with it between muh teeth!

[Ordis] "OPERATOR?

AM I DELICOUS?"

The only thing sweeter will be the sound of your silence, Ordis.

Thank-ya for watchin' and catch ya next time, Legion!

Take care!

For more infomation >> Warframe - How to Get Nidas Parts ASAP - Update 19.5 The Glast Gambit [1080HD] - Duration: 1:59.

-------------------------------------------

Undocumented youth brace for a frightening future under Trump - Duration: 2:40.

If your family was undocumented

They're bringing drugs, they're bringing crime they're rapists.

And some, I assume, are good people.

If I'm elected they're gonna be out day one.

Build that wall!

Build that wall!

Donald Trump has defeated Hillary Clinton to become president elect.

I was just...

I couldn't believe it.

It felt like a nightmare you know?

'Cause we didn't really think this could actually happen.

I had friends that were around me making jokes, saying 'oh are you going to go back to Mexico now?

I was just laughing on the outside but on the inside I was really scared, really nervous

for what's about to happen.

What do you do about the DACA order now?

The executive order gets rescinded.

They're here illegally, they're gonna have to go and they're gonna have to come

in legally.

We're gonna get rid of the bad ones, the bad ones are gonna be out of here fast, as

far as everybody else, we have to go through the process.

Should I go into hiding?

Am I not going to be able to continue working where I am working?

What am I going to do?

There aren't many answers so there is a lot of uncertainty.

My daughter is crying a lot.

I sit with her and I say, you don't worry.

You continue your dreams and always I help you.

I am a mother.

I don't know where did I get that strength, but I did.

I soon will be 18, and after that you're an adult so I'm not exactly sure what's

going to happen to me and how I will be protected.

But for right now I feel that I am okay, I'm safe.

I want to stay hopeful and I want to stay positive, I think that's the best that we

can do right now.

We have to wait until the president say something, you know?

I think that's...we have to be patient and keep believing in God and keep going

on in everyday life.

For more infomation >> Undocumented youth brace for a frightening future under Trump - Duration: 2:40.

-------------------------------------------

What the Bot? | This Isn't Coffee! - Duration: 1:17.

Whoa... Nice bike, Dude!

Yeah...

My Harley and I have been through a lot together.

You know...

Four wheels move the body, but two wheels move the soul.

Wow!

It takes more love to share the saddle than it does to share the bed.

Cool.

Bikes don't leak oil,

they mark their territory.

Uh huh...

I work to ride

and ride to work.

Of course you do.

Only bikers know why dogs stick their head out of a car window.

Right...

So... Can I get some coffee?

Oh yeah, sure.

Here you go.

Thanks!

This isn't coffee! It's oil!

What's the difference?

What the Bot?!

For more infomation >> What the Bot? | This Isn't Coffee! - Duration: 1:17.

-------------------------------------------

MICROSCOPE Turn Your Phone Into A DIGITAL Microscope ✔️ BEST Of HiHACKER - Duration: 3:34.

Microscope best digital microscope top 10 . When i was a kid.

I used to make different things out of cool trash, I mean cool things out of trash.

And microscope was one of them.

So i wanna share a short interesting story of mine.

I mean, story of this kung fu master from . This was before i heard about this wonderful

site called You Tube.

So don't you be thinking, i saw a video and replicated it.

My dad, had a samsung smartphone, which was coolest back then.

And i?

Removed it's back camera without telling anyone.

I Didn't just removed it, i took a knife, heated it, and made a hole on the back of

the phone.

And the funny thing is, my dad didn't noticed it for 3 days.

Maybe it's because, people didn't use to take photos of food.

Anyway, so i took the camera, removed it's lens, and saw a beautiful world, which was

tiny, i think.

I saw my own cells, my plant's cells, my dog cells, and many other amazing things.

Today I'm going to make the microscope again.

But this time it's digital.

You can try this yourself, you'll love it!

Things you gonna need is, a lens.

Like this one.

You can get it from a laser pointer.

Like this one right here, which worth less than a dollar, or you can get a lens out of

an old useless phone, or out of a tablet, if you have a spare one.

Once you have them.

Take your phone or tablet.

Place the lense above the camera.

You can place it on the front camera, so that you can see the screen as well.

And now take your stuff out, and observe this beautiful world.

What i made few years ago, was different than this one.

Because this is a simple digital microscope.

Or if you wanna call it that

For more infomation >> MICROSCOPE Turn Your Phone Into A DIGITAL Microscope ✔️ BEST Of HiHACKER - Duration: 3:34.

-------------------------------------------

How an Engine Cooling System Works and Operates - Also Diagnosis and Fix - Duration: 10:06.

- Hey, how's it going folks?

Today, I'm gonna show you how

your car's engine's cooling system works and operates

but not just that,

I'm also going to walk you through a diagnosis procedure

for a car with suspected cooling system issues.

Now, before we get started,

I would just like to mention

that if you see any tools or products

used in this video that peak your interest,

I put links to them in the description box.

So, don't be afraid to click on them and check them out.

Alright, so the job of your cooling system

is to first make sure that the temperature of your engine

does not rise beyond a certain point.

And the components that help it to achieve this

are going to be the radiator,

the radiator cooling fans

that could be electronically controlled like this one here,

or fans that are belt driven

like what we have on this Ford here,

And a belt driven water pump,

which, of course, is responsible

for circulating coolant throughout your cooling system.

Now, your cooling system also has a second function

which is to make sure

that your engine reaches a preset operating temperature

before it starts cooling it down.

And how it's able to do this is by using a thermostat,

which is what you see right here.

Alright, so we'll do a quick demonstration

on how everything works together on this whiteboard.

And, for the sake of argument,

we're gonna assume that we are going to start our engine

from a cold start.

Alright, so from a cold start

our thermostat is going to be in the closed position,

which is the position you see here.

In the closed position,

it does not allow for any coolant to pass through it.

So, here's our thermostat on this diagram

and when this is closed, it keeps coolant inside the engine.

See, inside your block you have these coolant passages

that are there so that coolant

can circulate through your cylinder block

and keep the cylinder block cool during normal operation.

And, of course you also have them inside your cylinder head

all around the combustion chambers as well.

So, the job of your thermostat is to remain closed

until the coolant inside your engine

reaches a preset temperature,

and then this valve opens

and allows for the circulation of coolant

throughout your cooling system.

And, once the thermostat opens,

your water pump is gonna pump coolant

through this upper radiator hose,

into your radiator.

And, once coolant gets inside your radiator,

it's cooled down by exchanging its heat

through the fins of the radiator.

And then, from there, once its cooled,

it goes back to the engine

and runs through the engine

and keeps it cool.

And from there, again, the whole cycle repeats itself.

Now, I should mention

that not always coolant flows

from the lower radiator hose through the engine

to the upper radiator hose.

Sometimes it goes the other way around.

And when it goes the other way around,

your thermostat is going to be right here

where your radiator hose goes to your engine,

keeping the coolant inside your engine.

As is going to be the case

on this 2000 Subaru Legacy with a 2.5 Liter engine.

So on this engine,

coolant flows from this one into the engine.

And once the coolant is properly warmed,

the thermostat will open

and then from there,

it will flow back through the radiator,

and the whole thing will repeat itself.

Alright, so that was simple enough,

but now let's go diagnose

and fix a car that I suspect has some cooling issues.

(car engine)

But before we do that, we gotta do something.

I'm sure you guys can probably guess just by listening,

what's about to happen.

(ice cream truck)

Yeah, thank you.

Yeah, ice cream.

And the vehicle that we are going to be working on

is going to be this 1996 Chevy truck

with a 5.7 liter V-8 gas engine.

And the issue we've been having with this truck,

is that the temperature gauge

does not rise after the car is supposedly warmed up.

So, we're gonna check the thermostat first,

because that's going to be the main culprit.

If that's not it,

then it would probably be

either the coolant temperature censor,

or the gauge in the dash.

But we're gonna start off with the thermostat.

Alright, so things are gonna be a little tight here,

but here's a look at our upper radiator hose.

And if we follow that, obviously,

that goes to our thermostat.

And all the way here on the other side,

on the lower side of the radiator, right down there,

if you can make it out, there is our lower radiator hose.

So, next we're gonna start our engine.

But, you wanna make sure you start with a cold engine.

(car engine)

Alright, so here's the temperature

for our upper radiator hose: 90 degrees.

And the temperature for our lower radiator hose:

just about the same.

Now, if our thermostat

and the cooling system is working properly,

the temperature for the radiator hose,

while the thermostat is closed,

should remain just about 90 degrees.

But, the temperature for the lower radiator hose,

should start to climb now.

Alright, so it's been a few minutes

since the engine has been running.

Let's recheck our temperatures.

Here is our upper radiator hose at 108.

That's interesting.

And here is our lower radiator hose at 108 also.

So, there is a clear example

of a thermostat that is stuck in the open position.

Thermostat is stuck open,

therefore, it doesn't allow

for your engine to properly warm up.

See, when it's stuck open at start-up,

the coolant just continuously flows

through your cooling system

and its temperature,

is not allowed to rise to the correct level.

And again, another sign that your thermostat is stuck open,

is you not being able to get any warm air inside the car.

And, look at our temperature gauge

after about seven minutes: stuck at pretty much hundred.

Alright, so there now that we have verified

that our thermostat is indeed stuck open,

we are gonna replace our thermostat.

Alright, in order to remove our thermostat,

we're gonna first start off

by removing our air filter housing.

And we're gonna do that be removing these two bolts here.

(drill squeal)

And then this one on the side.

And then we're gonna loosen this clamp

that is attaching it to our MAP sensor.

Alright, now we should be able to get it out of here.

Alright, next we'll go inside the car,

and then we need to obviously

remove the little piece that's covering our engine.

It's pretty easy.

There it is.

It's only held on there by four bolts,

two on the bottom and two on the sides,

and then there is, of course,

this little plastic cover that's sitting over it.

Then, using my long ratcheting wrenches,

I'm gonna remove this bolt that's holding in this air tube

that's going to the top of our throttle-body.

So now we can just lift up on this end

and then go to the front of the engine

and then on this end, we gonna first remove this connector

that goes to our MAP sensor

and then wiggle this

and we should be able to get it out of here.

There's gonna be another connector

for our inlet air temperate sensor

that's just behind our MAP sensor.

And also, a PCV hose that's on the side.

I'll show you in a bit.

But there, this is the PCV hose

that goes on the side that you need to remove.

Alright, so if you follow the upper radiator hose,

it goes around, loops around,

and there, is the look at our thermostat housing.

Looks like there are two studs

on this side and that side that are holding it in.

This one also has an extra nut here

that also holds a ground cable.

That's very important,

and you want to make sure you put that back

when you go to reinstall this.

And here's a look at a coolant temperature sensor.

That's just on here.

So we're gonna actually go with this one first.

Alright, so first this upper bolt.

And, there's the nut that's holding this ground wire in.

Now, before we remove those two nuts

that are holding our thermostat housing in,

it's a good idea, obviously, to drain

the coolant from your engine.

Alright, here's the rear one.

Alright, now we'll just pull this

and then let the rest of the coolant drain out.

There we go.

Alright, here's a look at our suspect.

I'm just gonna yank it out of here.

There we go.

And well what do you know?

It is stuck open.

You can see right through it, right there.

And here's a look at our new thermostat

and as you can see it is closed

and it only supposed to open

when it reaches a predetermined temperature,

which is 195 degrees for this thermostat.

Alright, so we'll put our new thermostat back in,

make sure its evenly seated where it's supposed to be.

And then I'm gonna spend some time

cleaning the mating surface on this end.

On the thermostat side it's not as important

because there's a little seat or a groove

where the seal goes inside.

(high pitched whine)

Alright, so putting it back together

is gonna be pretty much the reversal

of the removal from here.

So I'm not gonna get that on video,

but we'll cut to the, next we'll cut to where

we gonna do the temperature test

on our upper and lower radiator hoses.

Alright, here we go.

(engine starts)

Alright, a few minutes later,

this upper one is about 80 - 81.

This lower one, however, is now about 92 - 92 1/2.

Now the temperature of these hoses

is not obviously the temperature of the coolant.

For that, we gonna go inside the car

and check our temperature gauge.

Alright, as you can see,

it's only been a couple of minutes

but we're already up to about 150 according to our gauge.

So, things are definitely working proper right now.

Alright, so in the lower 80's.

And this bottom one is about 123 - 124,

and climbing steadily.

And our thermostat just opened, I believe,

now we're at 119 - 120 on this upper one.

And we are much cooler now on the lower one: about 98.

So that's about 20 degrees or so cooler.

Again, that's the temperature difference

between the two hoses.

I don't exactly know how much cooler the coolant is

when it passes through our radiator,

but it's obviously much cooler.

So that's it folks,

hope you enjoyed watching this video.

If you did though, do me a favor and share this video

on your favorite social network.

And also consider checking out these other related videos

of which I put links to

on this side of your screen that you can click on.

There will also be links

in the description box down below as well.

Alright, thanks for watching.

I'll see you guys next time.

(heavy rock music)

For more infomation >> How an Engine Cooling System Works and Operates - Also Diagnosis and Fix - Duration: 10:06.

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Top 7 des personnages de séries les plus insupportables - Duration: 4:00.

For more infomation >> Top 7 des personnages de séries les plus insupportables - Duration: 4:00.

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Neuron action potential - physiology - Duration: 10:25.

Neurons are the cells that make up our nervous system, and they're made up of three main

parts.

The dendrites, which are little branches off of the neuron that receive signals from other

neurons, the soma, or cell body, which has all of the neuron's main organelles like

the nucleus, and the axon which is intermittently wrapped in fatty myelin.

Those dendrites receive signals from other neurons via neurotransmitters, which when

they bind to receptors on the dendrite act as a chemical signal.

That binding opens ion channels that allow charged ions to flow in and out of the cell,

converting the chemical signal into an electrical signal.

Since a single neuron can have a ton of dendrites receiving input, if the combined effect of

multiple dendrites changes the overall charge of the cell enough, then it triggers an action

potential- which is an electrical signal that races down the axon up to 100 meters per second,

triggering the release of neurotransmitter on the other end and further relaying the

signal.

So neurons use neurotransmitters as a signal to communicate with each other, but they use

the action potential to propagate that signal within the cell.

Some of these neurons can be very long, especially ones that go from the spinal cord to the toes,

so the movement of this electrical signal is super important!

But why does the cell have an electric charge in the first place?

Well, it's based on the different concentrations of ions on the inside versus outside of the

cell.

Generally speaking, there are more Na+ or sodium ions, Cl- or chloride ions, and Ca2+

or calcium ions on the outside, and more K+ or potassium ion and A- which we just use

for negatively charged anions, on the inside.

Overall, the distribution of these ions gives the cell a net negative charge of close to

-65 millivolts relative to the outside environment - this is called the neuron's resting membrane

potential.

When a neurotransmitter binds to a receptor on the dendrite, a ligand-gated ion channel

opens up to allow certain ions to flow in, depending on the channel.

Ligand-gated literally means that the gate responds to a ligand, which in this case is

a neurotransmitter.

So if we take the example of a ligand-gated Na+ ion channel, which, when it opens, lets

Na+ flow into the cell.

The extra positive charge that flows in makes the cell less negative (since remember it's

usually -65mV), and therefore less "polar" - so that's why gaining positive charge

is called depolarization.

Neurotransmitters typically open various ligand-gated ion channels all at once, so ions like sodium

and calcium, may flow in, while other ions like potassium, may flow out, which would

actually mean some positive charge leaves the cell.

In the end though - when it's all added up - if there is a net influx of positive

charge, then it's called an excitatory postsynaptic potential (EPSP).

In contrast, the opening of only ligand-gated Cl- ion channels would cause a net influx

of negative charge, creating an inhibitory postsynaptic potential (IPSP), making the

cell potential more negative or repolarizing it.

Now, a single EPSP or IPSP causes only a small change on the resting membrane potential,

but, if there are enough EPSPs across multiple sites on the dendrites then collectively they

can push the membrane potential to a specific threshold value- typically about -55mV, although

this can vary by tissue.

When this occurs, it triggers the opening of voltage-gated Na+ channels at the start

of the axon - the axon hillock, voltage-gated channels open in response to a change in voltage,

and when these open sodium to rush into the cell.

The influx of sodium ions and the resulting change in membrane potential causes nearby

voltage-gated sodium channels to open up as well - setting off a chain reaction that continues

down the entire length of the axon—which is our action potential, and when this happens,

we say that the neuron has 'fired.'

Once a lot of sodium has rushed across the neuronal membrane, the call actually becomes

positively charged relative to the external environment - up to about +40mV.

The depolarization process ends when the sodium channel stops allowing sodium to flow into

the cells- a process known as inactivation.

But this state is different than when the channel's closed, or open for that matter,

which is what most of the other channels have.

The voltage-gated sodium channel, though, is unique in that it has what's known as

the inactivation gate, which blocks sodium influx shortly after depolarization, until

the cell repolarizes and the channel enters the closed state again and the inactivation

gate stops blocking influx, although even though the inactivation gate's not blocking,

the channel's still closed so no sodium enters the cell.

This middle open state therefore is the only state where sodium gets let into the cell

through the channel, and this is a very short window of time.

Now in addition to these sodium voltage-gated channels, we've also got potassium voltage-gated

channels, which are slow to respond and don't open until the sodium channels have already

opened and become inactivated.

The result is that after the initial sodium rush into the cell, potassium flows out of

the cell down its own electrochemical gradient- removing some positive charge and blunting

the effect of the sodium depolarization.

The potassium channels, do not have a separate inactivation gate and therefore remain open

for slightly longer, which means that there is a period of time when there is a net movement

of positive ions out of the cell, causing the membrane potential to become more negative,

or repolarize.

During this repolarization phase, the cell also relies on the sodium-potassium pump,

an active transporter that moves three sodiums out of the cell and two potassiums into it.

It's during this repolarization phase that the cell's in its absolute refractory period,

since the sodium channels are inactivated and won't respond to any amount of stimuli.

This absolute refractory period keeps the action potentials from happening too close

together in time, but also keeps the action potential moving in one direction.

The combined efforts of this pump and the extended opening of the potassium channels

results in a small period of overcorrection where the neuron becomes hyperpolarized relative

to the resting potential, and at this point the sodium channels go back to their initial

closed state, and for a short period the potassium channels stay open.

Now we're in the relative refractory period since the sodium channels are closed but can

be activated, but because the potassium channels are still open and we're in a hyperpolarized

state, so it's takes a strong stimulus to do so.

Finally, as the potassium channels close, the neuron returns to it's resting membrane

potential.

Alright, as a quick graphical recap, with membrane potential on the y and time on the

x.

First we start at resting potential of around -65 mV and voltage-gated sodium and potassium

channels are closed, we receive EPSPs enough to hit threshold at about -55 mV, voltage-gated

sodium channels open and we reach a peak of about +40 mV, at which point the sodium channels

become inactivated and we're in the absolute refractory period.

Voltage-gated potassium channels open, and along with the sodium-potassium pump, start

to repolarize the cell, so much so that it overshoots and hyperpolarizes the cell.

Next the sodium channels enter their closed resting state as potassium channels start

to close we're in the relative refractory period, until finally they all close and we

reach our resting membrane potential.

Alright, so this process of positive sodium ions moving in and depolarizing the cell transmits

the electrical signal down the length of the axon.

Great.

But really, this process isn't that fast.

So that's where the fatty myelin comes in, which comes from glial cells like Schwann

cells or oligodendrocytes.

These myelinated areas don't have voltage-gated ion channels spanning the membrane, so ions

can't simply flow into the cell, that only happens in the spots between the myelin, called

nodes of Ranvier.

So instead of propagating via channels, the charge essentially jumps from node to node.

That said though, these ions aren't just diffusing down the length of the myelin to

the other side...that'd be way to slow.

What actually happens is more like the sodium ions rushing in bumps other positive sodium

ions already inside the cell, which bumps another one, and so on until it reaches the

next node.

The charge moving in this way with the myelinated areas moves really fast, and is called saltatory

conduction, which makes it look like the action potential "jumps" from one one node to

the next.

Okay extremely quick recap - neuron action potentials happen when dendrites receive enough

EPSPs to open voltage-gated sodium channels, which cause rapid depolarization of the neuronal

membrane and propagation of an electrical charge from node to node down the length of

the axon.

Thanks for watching, you can help support us by donating on patreon, or subscribing

to our channel, or telling your friends about us on social media

For more infomation >> Neuron action potential - physiology - Duration: 10:25.

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What Happens If I Get a DUI While Visiting Colorado? - Duration: 1:18.

Well anybody who's been charged with a marijuana dui at the very least should come in and have

a consultation.

We can look at your blood test results, we can talk to you about how long you've used

marijuana, how often you use marijuana and from there we can start talking about how

viable of a defense you might have.

You come into Colorado for one reason or another and you find out, you find yourself being

prosecuted for dui.

You're going to have to have a Colorado lawyer.

If you're from some other state you're not going to be able to hire an attorney from

your home state to come here unless they happen to be licensed in Colorado.

We see it all the time, people come in to ski, maybe they enjoy a beer, maybe they enjoy

some marijuana.

They're driving back to their hotel, they get pulled over and then they had to leave

the next day.

Well, you have to take care of it and you have to take care of it, number one, because

there will be a warrant put out for your arrest if you don't, there's also going to be ramifications

on your driver's license.

Colorado will tell your home state that you have a DUI here and so it could suspend your

driver's license in your home state as well.

For more information, go to our website at DUIDefenseMatters.com.

For more infomation >> What Happens If I Get a DUI While Visiting Colorado? - Duration: 1:18.

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Does The Canon M5 Have a Microphone Input? | Canon EOS M5 FAQ Video - Duration: 1:26.

Hello again!

Izzy at Digital Goja showrooms and here is one of the most

frequently asked questions about the EOS M5.

If this video helps you out remember hit me up with a like button

underneath and subscribe to the channel for future frequently asked questions

such as this and to share with fellow photographers and videographers and

don't forget when in Miami visit Digital Goja showrooms.

Digital Goja showrooms has one of the nation's largest in-store

selection of authorized Canon cameras and lenses.

Does it have a microphone input?

Yes it does, it does have built in mono microphone connection but honestly if

you're gonna do professional audio you want to use an external mic.

So notice how here we have the 3.5 port so that

way you can connect your external microphone whether it's a condenser or

lavalier.

They did not give you a headphone jack,

they only gave you a microphone jack.

So if you're looking for a headphone jack it does not have one but it does

have for external microphone input.

For more infomation >> Does The Canon M5 Have a Microphone Input? | Canon EOS M5 FAQ Video - Duration: 1:26.

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What Does The Bible Say About Christian Tithing | GotQuestons.org - Duration: 2:25.

Question: "What does the Bible say about Christian tithing?

Should a Christian tithe?"

In this week's video, we'll see what the Bible has to say,

and at the end I'll point you to some helpful resources, so stick around for that.

Many Christians struggle with the issue of tithing.

In some churches giving is over-emphasized.

At the same time, many Christians refuse to submit to the biblical exhortations about

making offerings to the Lord.

Tithing and giving is intended to be a joy and a blessing.

Sadly, that is sometimes not the case in the church today.

Tithing is an Old Testament concept.

The tithe was a requirement of the Law in which the Israelites were to give 10 percent

of the crops they grew and the livestock they raised to the tabernacle/temple.

In fact, the Old Testament Law required multiple tithes—one for the Levites, one for the

use of the temple and the feasts, and one for the poor of the land—which would have

pushed the total to around 23.3 percent.

Some understand the Old Testament tithe as a method of taxation to provide for the needs

of the priests and Levites in the sacrificial system.

After the death of Jesus Christ fulfilled the Law, the New Testament nowhere commands,

or even recommends, that Christians submit to a legalistic tithe system.

The New Testament nowhere designates a percentage of income a person should set aside, but only

says gifts should be "in keeping with income."

Some in the Christian church have taken the 10 percent figure from the Old Testament tithe

and applied it as a "recommended minimum" for Christians in their giving.

The New Testament talks about the importance and benefits of giving.

We are to give as we are able.

Sometimes that means giving more than 10 percent; sometimes that may mean giving less.

It all depends on the ability of the Christian and the needs of the body of Christ.

Every Christian should diligently pray and seek God's wisdom in the matter of participating

in tithing and/or how much to give.

Above all, all tithes and offerings should be given with pure motives and an attitude

of worship to God and service to the body of Christ.

"Each man should give what he has decided in his heart to give, not reluctantly or under

compulsion, for God loves a cheerful giver."

Hungry for more?

Subscribe so you don't miss the next video!

Visit GotQuestions.org for more great content.

And check out the details section below this video: There's a book I highly recommend,

along with other articles that will help you better understand what the Bible says about tithing.

If you'd like to learn about Bible Munch, or if you're interested in Bite-sized devotionals,

subscribe to Bible Munch on YouTube.

It's linked right here.

Remember, GotQuestions?

The Bible has answers!

We'll help you find them!

For more infomation >> What Does The Bible Say About Christian Tithing | GotQuestons.org - Duration: 2:25.

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Ice Road Truck Driving Race | Racing Action & Adventure | Videos Kids Games - Duration: 11:04.

Ice Road Truck Driving Race | Racing Action & Adventure | Videos Kids Games

Thank you for watching! Educational Kids Games.

For more infomation >> Ice Road Truck Driving Race | Racing Action & Adventure | Videos Kids Games - Duration: 11:04.

-------------------------------------------

Train Crash Compilation 2016 - Train Accidents and Crash Compilation Videos HD - Duration: 12:34.

Train Crash Compilation 2016 - Train Accidents and Crash Compilation Videos HD

Train Crash Long Duration Compilation Videos Train crash video contains documentary or educational context which can help the viewer about to understand the consequence of unacceptable driving behavior. Be careful on the road. Drive safely and keep yourself and others safe.

For more infomation >> Train Crash Compilation 2016 - Train Accidents and Crash Compilation Videos HD - Duration: 12:34.

-------------------------------------------

WEIRD CLOUD FELL FROM THE SKY!!!! - Duration: 3:34.

WEIRD CLOUD FELL FROM THE SKY!!!!

A weird cloud seems to be falling from the sky, or at least allegedly, have a closer look with us and let's see what we can find out! Paranormal videos. Weird Videos Caught on Tape.

For more infomation >> WEIRD CLOUD FELL FROM THE SKY!!!! - Duration: 3:34.

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how to share hostpot using connectify me / sharing wifi hostpot from laptop - Duration: 4:00.

hii

open browser

go to google

connectify or connectify me

you can copy link from description also

download it

you can download from any sites

installing !!

you can reboot now also ...

see there

asking for reboot ..

this is wifi name

and this is password for wifi

then you can start now

wait few second !!

ok hostpot is shared sucessfully !!!

you can stop from here .

thank you

thanx

For more infomation >> how to share hostpot using connectify me / sharing wifi hostpot from laptop - Duration: 4:00.

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Top 10 Real Alien Sightings Caught on Camera 2016 - Real Alien Sightings in the World Caught on Tape - Duration: 14:43.

Top 10 Real Alien Sightings Caught on Camera 2016 - Real Alien Sightings in the World Caught on Tape

This video is covered under fair use and does not breach copyright and everything has only been used in parts and only to get my point across and everything has been added to or adjusted, however if you have any queries please message me first and it can be sorted out very easily!

For more infomation >> Top 10 Real Alien Sightings Caught on Camera 2016 - Real Alien Sightings in the World Caught on Tape - Duration: 14:43.

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Giant Snake In Toilet Attacks Spatula Girl Victoria And Annabelle Toy Freaks Family | Creactor Funny - Duration: 2:08.

nice we g2g o.o what wash who snack

oh lame oh she heard all scream thing

Oh toilet but what is this thang

oh shit look back

oh oh alright it's great you get it

only there's an and understand that

oh yes

yo-o-o-o-o-m has top

oh oh oh yeah chief

For more infomation >> Giant Snake In Toilet Attacks Spatula Girl Victoria And Annabelle Toy Freaks Family | Creactor Funny - Duration: 2:08.

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RomanAtwoodVlogs: WE CAUGHT SANTA!! My New Prank !! RomanAtwood - Duration: 12:46.

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